精彩!!绝对的精华帖~ ipsvirus 以后可以多写些类似风格的文章,貌似以前一位大牛有过这种风格 |
写的非常不错啊 |
rojjer 发表于 2015-8-19 13:22 |
只能说你很真的很牛B。 |
精品 |
必须赞! 我们曾经的VIP课题(Viral Ion channel Project)! |
写的非常不错啊 |
厉害啊 |
精华啊,佩服 |
师兄好博学,以武侠小说的style描写了学术界的八卦,最后还歌颂了我们亲爱的中国病毒学论坛,此处必须有掌声啊。。。。 |
参考文献 [1] J.R. St-Jean, H. Jacomy, M.Desforges, A. Vabret, F. Freymuth, P.J. Talbot, Human respiratory coronavirusOC43: genetic stability and neuroinvasion, J Virol, 78 (2004) 8824-8834. [2] L. Vijgen, E. Keyaerts, P.Lemey, E. Moës, S. Li, A.-M. Vandamme, M. Van Ranst, Circulation of geneticallydistinct contemporary human coronavirus OC43 strains, Virology, 337 (2005)85-92. [3] L. Vijgen, P. Lemey, E.Keyaerts, M. Van Ranst, Genetic variability of human respiratory coronavirusOC43, J Virol, 79 (2005) 3223-3224; author reply 3224-3225. [4] C. Lin, B.D. Lindenbach, B.M.Pragai, D.W. McCourt, C.M. Rice, Processing in the hepatitis C virus E2-NS2region: identification of p7 and two distinct E2-specific products withdifferent C termini, J Virol, 68 (1994) 5063-5073. [5] S.D.C. Griffin, L.P. Beales,D.S. Clarke, O. Worsfold, S.D. Evans, J. Jaeger, M.P.G. Harris, D.J. Rowlands,The p7 protein of hepatitis C virus forms an ion channel that is blocked by theantiviral drug, Amantadine, FEBS Letters, 535 (2003) 34-38. [6] E. Steinmann, F. Penin, S.Kallis, A.H. Patel, R. Bartenschlager, T. Pietschmann, Hepatitis C virus p7protein is crucial for assembly and release of infectious virions, PLoSPathogens, 3 (2007) e103. [7] E. Steinmann, T. Whitfield, S.Kallis, R.A. Dwek, N. Zitzmann, T. Pietschmann, R. Bartenschlager, Antiviraleffects of amantadine and iminosugar derivatives against hepatitis C virus,Hepatology, 46 (2007) 330-338. [8] S. Griffin, C. Stgelais, A.M.Owsianka, A.H. Patel, D. Rowlands, M. Harris, Genotype-dependent sensitivity ofhepatitis C virus to inhibitors of the p7 ion channel, Hepatology, 48 (2008)1779-1790. [9] B.P. Doehle, K. Chang, A.Rustagi, J. McNevin, M.J. McElrath, M. Gale, Jr., Vpu mediates depletion ofinterferon regulatory factor 3 during HIV infection by a lysosome-dependentmechanism, J Virol, 86 (2012) 8367-8374. [10] B.P. Doehle, K. Chang, L.Fleming, J. McNevin, F. Hladik, M.J. McElrath, M. Gale, Jr., Vpu-deficient HIVstrains stimulate innate immune signaling responses in target cells, J Virol,86 (2012) 8499-8506. [11] D. Hotter, F. Kirchhoff, D.Sauter, HIV-1 Vpu Does Not Degrade Interferon Regulatory Factor 3, Journal ofVirology, 87 (2013) 7160-7165. [12] S.Y. Park, A.A. Waheed, Z.R.Zhang, E.O. Freed, J.S. Bonifacino, HIV-1 Vpu accessory protein inducescaspase-mediated cleavage of IRF3 transcription factor, J Biol Chem, 289 (2014)35102-35110. [13] L. Manganaro, E. de Castro,A.M. Maestre, K. Olivieri, A. García-Sastre, A. Fernandez-Sesma, V. Simon, HIVVpu interferes with NF-κB activity but not with Interferon Regulatory Factor 3,Journal of Virology, (2015) JVI.01596-01515. |
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